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hna rabbit  (Cell Signaling Technology Inc)


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    Cell Signaling Technology Inc hna rabbit
    Hna Rabbit, supplied by Cell Signaling Technology Inc, used in various techniques. Bioz Stars score: 96/100, based on 3049 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
    https://www.bioz.com/result/hna rabbit/product/Cell Signaling Technology Inc
    Average 96 stars, based on 3049 article reviews
    hna rabbit - by Bioz Stars, 2026-06
    96/100 stars

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    Effect of 100 μM rufinamide on human Na v channel isoforms involved in epilepsy. (A) Molecular organization of rufinamide consisting of the 1,2,3-triazole ring connecting the 2,6-difluorophenyl and the carboxamide. (B–E) The left column shows G / G max and I / I max relationships, whereas the right column displays the recovery from fast inactivation. The figure shows that rufinamide inhibits hNa v 1.1 opening whereas the effect on hNa v 1.6 is not significant compared to the effect of DMSO treatment of this particular isoform (see Table ). Furthermore, the recovery from fast inactivation slows for the four Na v channel isoforms tested here. All data are shown before (green, DMSO control) and after (red) addition of 100 μM rufinamide to hNa v 1.1 (B), hNa v 1.2 (C), hNa v 1.3 (D), and hNa v 1.6 (E). All fit values are listed in Table . n = 5–8, and error bars represent the standard error of the mean.

    Journal: ACS Chemical Biology

    Article Title: Na v 1.1 Modulation by a Novel Triazole Compound Attenuates Epileptic Seizures in Rodents

    doi: 10.1021/cb500108p

    Figure Lengend Snippet: Effect of 100 μM rufinamide on human Na v channel isoforms involved in epilepsy. (A) Molecular organization of rufinamide consisting of the 1,2,3-triazole ring connecting the 2,6-difluorophenyl and the carboxamide. (B–E) The left column shows G / G max and I / I max relationships, whereas the right column displays the recovery from fast inactivation. The figure shows that rufinamide inhibits hNa v 1.1 opening whereas the effect on hNa v 1.6 is not significant compared to the effect of DMSO treatment of this particular isoform (see Table ). Furthermore, the recovery from fast inactivation slows for the four Na v channel isoforms tested here. All data are shown before (green, DMSO control) and after (red) addition of 100 μM rufinamide to hNa v 1.1 (B), hNa v 1.2 (C), hNa v 1.3 (D), and hNa v 1.6 (E). All fit values are listed in Table . n = 5–8, and error bars represent the standard error of the mean.

    Article Snippet: Human (h) hNa v 1.1 (NM_001165963.1), hNa v 1.2 (NM_021007.2), hNa v 1.3 (NM_006922.3), hNa v 1.6 (NM_014191.3), and hβ 1 (NM_001037.4) clones were obtained from OriGene Technologies, Inc., and modified with an ER forwarding motif and the rNa v 1.2a 3′UTR region for expression in Xenopus laevis oocytes (acquired from Xenopus One).

    Techniques:

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    Journal: ACS Chemical Biology

    Article Title: Na v 1.1 Modulation by a Novel Triazole Compound Attenuates Epileptic Seizures in Rodents

    doi: 10.1021/cb500108p

    Figure Lengend Snippet: Effects of 100 μM Rufinamide and Its Derivatives on Human Na v Channel Isoforms a

    Article Snippet: Human (h) hNa v 1.1 (NM_001165963.1), hNa v 1.2 (NM_021007.2), hNa v 1.3 (NM_006922.3), hNa v 1.6 (NM_014191.3), and hβ 1 (NM_001037.4) clones were obtained from OriGene Technologies, Inc., and modified with an ER forwarding motif and the rNa v 1.2a 3′UTR region for expression in Xenopus laevis oocytes (acquired from Xenopus One).

    Techniques: Activation Assay